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. 2023 Aug 18;14:1229724. doi: 10.3389/fimmu.2023.1229724

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Copyright © 2023 Strother, Brewer-Jensen, Becker-Dreps, Zepeda, May, Gonzalez, Reyes, McElvany, Averill, Mallory, Montmayeur, Costantini, Vinjé, Baric, Bucardo, Lindesmith and Diehl

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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GII.17 blockade antibody responses in young children are genotype-specific. (A) The fold change between blockade antibody titers before and after GII.17 infection was determined for GII.17, GI.3, GII.2, GII.4 Sydney, GII.6, and GII.12. For each donor, VLP for the GII.17 cluster with the highest post-infection blockade titer was used in the assay. Marker, donor; bar, GMT; error bars, 95% confidence intervals; dashed line, no change in titer; **, P < 0.001 relative to heterotypic responses by Dunn’s multiple comparisons test. (B) Correlation analysis of GII.17 titer compared with titers to other genotypes: Spearman correlation, P > 0.05 comparing GII.17 titers to those for each heterotypic genotype. Dashed line, limit of detection.