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. 2023 Aug 31;28(1):2251237. doi: 10.1080/13510002.2023.2251237

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© 2023 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. The terms on which this article has been published allow the posting of the Accepted Manuscript in a repository by the author(s) or with their consent.

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Figure 6. — Treatment with LIPUS inhibited ferroptosis-related oxidative stress in the ADR-induced CKD rat. (A) Representative micrographs of reactive oxygen species (ROS) staining in each group. Scale bar, 50 μm. (B) The absolute expression level of ROS was quantified as the average fluorescence intensity. The levels of lipid peroxide (LPO) (C), superoxide anionic (O₂^-) (D) and malondialdehyde (MDA) (E) in rat kidney tissue were assayed by enzyme linked immunosorbent assay (ELISA). Data represent the mean ± standard error of the mean (SEM) for 6 rats in each group. *p < 0.05, ** p < 0.01, ***p < 0.001, **** p < 0.0001.