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. 2023 Aug 1;55(8):1783–1794. doi: 10.1038/s12276-023-01065-2

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a Representative western blot results. Ctrl, vehicle-exposed control mice; VPA, VPA-exposed mice. b VPA-exposed mice showed significantly increased Rnf146 levels compared to control mice (n = 14 mice for control, n = 13 mice for VPA, two-tailed unpaired t test; t₂₅ = 2.283, *P = 0.0312). Rnf146 expression levels were normalized to tubulin levels. c VPA-exposed mice showed significantly decreased p-β-catenin levels, which were normalized to β-catenin levels, compared to control mice (n = 14 mice for control, n = 13 mice for VPA, two-tailed unpaired t test; t₂₅ = 2.519, *P = 0.0185). d VPA-exposed mice showed comparable β-catenin levels, which were normalized to tubulin levels, when compared to control mice (n = 14 mice for control, n = 13 mice for VPA, two-tailed unpaired t test; t₂₅ = 1.456, P = 0.1577).