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. 2023 Sep 6;14(9):594. doi: 10.1038/s41419-023-06116-0

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 8 — 20C binds to the core toxic β-sheet structure of α-Syn fibrils, decrease the content of β-sheet structure of α-Syn, thus inhibit the accumulation of α-Syn, further inhibit the maturation of α-Syn, improve the dysfunction of mitochondria, maintain the homeostasis of mitochondrial dynamics, protect dopamine neurons, and play a biological role in the treatment of PD.