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Genetic or pharmacological activation of TRPML1 ameliorates liver disease of PiZ mice
Graphic representation of therapeutic efficacy of TRPML1-mediated lysosomal exocytosis in PiZ mice. Delivery of ATZ polymers to lysosome for exocytosis may rely on ER-to-lysosome-associated degradation pathway (ERLAD) (1) or autophagosome-lysosome fusion (2).
