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. 2023 Jan 12;41(9):1296–1306. doi: 10.1038/s41587-022-01626-2

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Extended Data Fig. 4 — a, Comparison of the cytotoxicities of an anti-B7-H3 antibody (B7-H3-IgG1) and of an anti-B7-H3 NKCE molecule co-engaging NKp46 and CD16a (B7-H3-NKCE). CD64⁻negative A375 and LN229 cells, and CD64-positive THP-1 cells were used as the targets and purified resting NK cells as effectors. The data shown are representative of three independent experiments. b, Phenotype of the target cells used in a showing the expression of B7-H3, CD32a/b, CD64 and CD123 by flow cytometry.