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. 2023 Jun 2;30(9):1234–1248. doi: 10.1038/s41417-023-00633-y

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — A, B Venn diagram of genes positively correlated with LBH in the cancer types indicated (left). The identified common genes from multi-cancer analysis were used for PANTHER (middle) or KEGG (right) pathway analysis. Pathways that have at least 10 predicted common genes are listed. A Analysis of LBH positively correlated genes in three cancer types: COAD, STAD, and LIHC; or B, in a four-cancer combination: COAD, PAAD, LIHC, and ESCA. Note that the top three pathways are WNT, Integrin, and inflammation signaling throughout different cancer-type combinations. COAD; colon adenocarcinoma, ESCA; esophageal carcinoma, LIHC liver hepatocellular carcinoma, PAAD pancreatic adenocarcinoma, STAD stomach adenocarcinoma. See also Fig. S4 and Table S5.