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. 2023 Sep 15;14:174. doi: 10.1007/s12672-023-00784-2

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 8 — DDIT4 promotes the progression of lung cancer. A, B The mRNA expression of DDIT4 in A549 and PC9 cells transfected with siRNAs (siDDIT4) or siRNA control (siCon) was measured by qRT-PCR. Means ± SD are shown. ***p < 0.01 by unpaired Student’s t-test. C, D Downregulation of DDIT4 slowed down A549 and PC9 cell proliferation. E siDDIT4 significantly inhibited wound closure compared to the corresponding controls. F Migratory and invasive cells were dramatically reduced in A549 and PC9 cells transfected with siDDIT4