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. 2023 Sep 18;15:72. doi: 10.1186/s13073-023-01218-y

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — Intercellular interaction analysis in the context of intra-sample heterogeneity. A Intercellular interaction analysis in the immune-rich tumor region (cancer-immune) reveals activation of PAX5. B, C FOS and JUN are the active networks from the interaction analysis at the immune-poor tumor region (HCC-CAF). D PAX5 activation co-localizes with the expression of B cell markers concentrated at the borders of the immune-rich tumor region. E FOS and JUN networks are co-expressed with CAF markers adjacent to the immune-poor tumor region