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. 2023 Jan 28;195(10):5881–5902. doi: 10.1007/s12010-022-04292-x

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — (1) Sections in control no infected mice with normal hepatic lobular architecture ((H&E stain and Picro-Sirius red stains, × 100). (2) Section in control non-treated liver tissue showing normal hepatic architecture with no excess deposition of fibrous tissue (H&E stain and Picro-Sirius red stains, × 100). (3) Section in liver of group (2) showing deposition of fibrous tissue, mostly in the portal tract (H&E stain and Picro-Sirius red stains, × 200). (4) Sections in liver of group (3 and 4) showing deposition of variable amounts of fibrous tissue in hepatic parenchyma and the portal tract (H&E stain and Picro-Sirius red stains, × 200). (5) Liver section in group (5) showing nearly preserved hepatic lobular architecture, with no evidence of fibrosis or nodular transformation (H&E stain and Picro-Sirius red × 100). (6) Liver section in group 6 showing nearly preserved hepatic lobular architecture, with no evidence of fibrosis or nodular transformation (H&E stain, Picro-Sirius red X100). No other significant histopathological changes were detected within different groups concerning hepatocytic degeneration or lobular inflammation