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. 2023 Sep 21;11(9):e007110. doi: 10.1136/jitc-2023-007110

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© Author(s) (or their employer(s)) 2023. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ.

This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See http://creativecommons.org/licenses/by-nc/4.0/.

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PMN-MDSCs from tumor bearing mice enable A1207 tumor growth. Ly6G+ cells were harvested from spleens of C57BL/6 mice without tumors or bearing A1206 tumors on day 0, 5, 11 and 16. 0.5 million A1207+1.5 million Ly6G+ cells were transplanted to the flanks of recipient C57BL/6 mice on day 0. Initiated tumors were transplanted with another 1.5 million Ly6G+ cells on days 5, 11 and 16. Rate of complete A1207 tumor clearance was monitored over time. *p<0.05 determined by log-rank test. PMN-MDSCs; polymorphonuclear myeloid-derived suppressor cells; WT, wild type.