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. 2023 Sep 25;13:16010. doi: 10.1038/s41598-023-42550-y

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© The Author(s) 2023, corrected publication 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Photomicrographs of H&E-stained heart sections of various experimental groups. (a) Heart of control rat showing normal orientation ad striation of cardia muscles’ fibers (MFs). (b–d) Heart of obese rat showing marked degeneration (arrow), loss of striation, and scattered eosinophilia (short arrow) of the myofibers with increase intermuscular fat (dotted arrow). (d) The coronary vessels showing vacuolization of medial muscles (arrow) and focal fibroid necrosis (dotted arrow), and inflammatory cells infiltration (IF). (e, f) Heart of low dose ZnONPs administrated rat showing moderate degree of myofibers degeneration (arrow), mild eosinophilia (short arrow) with some intermuscular fat (dotted arrow). (g, h) Heart of high dose ZnONPs administrated rat showing good restoration of the cardiac muscle fibers with mild degeneration (arrow) and decreased intermuscular fat.