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Golgi-stain of hippocampal neurons. Prior work has shown that eFSE causes attenuation of CA1 apical dendrites (a), overgrowth of DG dendritic arbors, and maintenance of immature DG basal dendrites. (b) This raises the question as to whether eFSE fundamentally alters the biophysics of dendritic current oscillations and the frequency of signal coding mechanisms at entorhinal-hippocampal synaptic inputs along both the CA1 and DG somatodendritic axes.
