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. 2023 Sep 13;14:1218015. doi: 10.3389/fphar.2023.1218015

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Copyright © 2023 Huang, Xie, He, Song and Cao.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Anti-inflammatory mechanism of chlorogenic acid compounds.:promote; :inhibit; CGA:chlorogenic acid; VCAM-1:vascular cell adhesion molecule 1; MDA:malonaldehyde; NADPH:nicotinamide adenine dinucleotide phosphate; ROS:reactive oxygen species; TLR4:toll like receptor 4; MyD88:myeloid differentiation primary response gene (88); P-P38:phosphorylated P38 kinase; P-PERK1:phosphorylated extracellular signal-regulated protein kinase-1; P-PERK2:phosphorylated extracellular signal-regulated protein kinase-2; p-JNK:phosphorylated c-Jun amino-terminal kinase.

Inline graphic — Anti-inflammatory mechanism of chlorogenic acid compounds.:promote; :inhibit; CGA:chlorogenic acid; VCAM-1:vascular cell adhesion molecule 1; MDA:malonaldehyde; NADPH:nicotinamide adenine dinucleotide phosphate; ROS:reactive oxygen species; TLR4:toll like receptor 4; MyD88:myeloid differentiation primary response gene (88); P-P38:phosphorylated P38 kinase; P-PERK1:phosphorylated extracellular signal-regulated protein kinase-1; P-PERK2:phosphorylated extracellular signal-regulated protein kinase-2; p-JNK:phosphorylated c-Jun amino-terminal kinase.