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. 1998 Feb;42(2):269–276. doi: 10.1128/aac.42.2.269

TABLE 5.

Simultaneous determination of resistance to HIV-1 PR and RT inhibitors by recombinant viruses with plasma-derived sequences encoding PR and RTa

Drug IC50 (μM) (fold resistanceb)
Sample 1 Sample 2 Sample 3 Sample 4 Sample 5
AZT 0.32 (5) 66.6 (998) 5.60 (57) 73.6 (460) 31.9 (426)
3TC 7.77 (1) 35.7 (5) >100 (>13) >100 (>11) 18.42 (4)
ddI 2.34 (1) 10.1 (1) 12.4 (2) 1.98 (1) 7.62 (1)
ddC 1.94 (1) 3.52 (1) 9.75 (3) 9.21 (3) 2.06 (1)
d4T 1.95 (1) 7.68 (1) 7.21 (1) 7.73 (2) 7.15 (4)
Loviride 0.047 (1) 0.09 (1) 3.49 (46) 0.02 (1) >100 (>1700)
Nevirapine ND 0.32 (2) 94.7 (622) ND >100 (>758)
Indinavir 0.023 (3) 0.11 (7) 0.39 (21) 0.63 (8) 1.95 (104)
Saquinavir 0.046 (15) 0.003 (1) 0.05 (8) 0.36 (37) 3.50 (579)
Ritonavir 0.092 (4) 1.15 (23) 7.71 (120) 1.71 (20) 11.3 (175)
 Viral loadc 26,000 191,000 269,000 400,000 NA
 CD4 count    224      75      54     314 NA
a

The combined PR-RT resistance pattern (10 drugs) was determined for different samples. The results for five samples with different patterns of resistance to all inhibitors are presented. Resistance to nevirapine was not determined (ND) for samples 1 and 4. Results are expressed as IC50s and fold resistance. Viral load data and CD4 count (numbers of cells per milliliter) were not available (NA) for sample 5. 

b

Fold-increase in the mean IC50 relative to the mean IC50 for the wild type. The mean IC50s for both patient and wild-type viruses were derived from two to four separate susceptibility determinations by the MT4-MTT assay. 

c

Number of HIV-1 RNA copies per milliliter of plasma as determined by the Roche Amplicor HIV-1 Monitor assay.