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. 2023 Aug 28;64(10):100437. doi: 10.1016/j.jlr.2023.100437

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© 2023 The Authors

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 3 — Dysosmobacter welbionis J115^T–induced lipid changes in the brown adipose tissue. (A) Principal component analysis showing the distribution of individuals based on their brown adipose tissue lipid profile. (B) Volcano plot with the bioactive lipid fold change in the brown adipose tissue. (C) Changes in 15d-PGJ2. (D) Changes in 11,12-EET. (E) mRNA expression of PPARg related genes relative to the control group. (F) Regularized canonical correlation analysis including metabolic parameters, mRNA relative expression, and lipids in the brown adipose tissue. Number of mice per group: 9–10. Data were analyzed using one-way ANOVA followed by Tukey’s post hoc test for figure parts C–E. ∗P < 0.05; ∗∗P < 0.01; and ∗∗∗P < 0.001. Results are represented as mean ± SEM for figure parts C–E. 15d-PGJ₂, 15-deoxy-Δ12,14-prostaglandin J2; EET, epoxyeicosatrienoic acid; PPAR, peroxisome proliferator–activated receptor.