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. 2023 Aug 28;64(10):100437. doi: 10.1016/j.jlr.2023.100437

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© 2023 The Authors

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 5 — Dysosmobacter welbionis J115^T–induced lipid changes in the blood. (A) Principal component analysis showing the distribution of individuals based on their plasma lipid profile. (B) Changes in PGD2. (C) Changes in 15d-PGJ2. (D) Changes in 17-HDoHE. (E) Changes in C18-2OH. (F) Changes in C14-3OH. (G) Changes in C16-3OH. (H) Changes in C18-3OH. (I) Changes in 14,15-EET. (J) Changes in 12-HETE. (K) Changes in 9-oxoODE. (L) changes in 13-oxoODE. (M) Changes in 9-HODE. (N) Changes in 10-HODE. Statistical comparisons are either HFD versus CT or HFD+J115 versus CT. Number of mice per group: 8–10. Data were analyzed using one-way ANOVA followed by Tukey’s post hoc test ∗P < 0.05; ∗∗P < 0.01; ∗∗∗P < 0.001, and ∗∗∗∗P < 0.0001. Results are represented as mean ± SEM. 15d-PGJ₂, 15-deoxy-Δ12,14-prostaglandin J2; EET, epoxyeicosatrienoic acid; HDoHE, hydroxydocosahexaenoic acid; HFD, high-fat diet; oxoODE, hydroxyoctadecadienoic acid; PGD2, prostaglandin D2.