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. 2023 Oct 2;7(10):e0277. doi: 10.1097/HC9.0000000000000277

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Copyright © 2023 The Author(s). Published by Wolters Kluwer Health, Inc. on behalf of the American Association for the Study of Liver Diseases.

This is an open access article distributed under the terms of the Creative Commons Attribution-Non Commercial-No Derivatives License 4.0 (CCBY-NC-ND), where it is permissible to download and share the work provided it is properly cited. The work cannot be changed in any way or used commercially without permission from the journal. http://creativecommons.org/licenses/by-nc-nd/4.0/

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Immunohistochemistry evaluation of KDM6B and G0S2 expression. (A) HE staining and immunohistochemical analysis of KDM6B expression in HCC in representative KDM6B-high and KDM6B-low tissues. (B) Immunohistochemical staining of G0S2 in human HCC tissues. Low G0S2 expression was correlated with low KDM6B expression (p = 0.036). Magnification: ×100; scale bar: 100 μm. (C) Schematic illustration of the regulation mechanism of intracellular lipid by KDM6B-G0S2-ATGL/PNPLA2 pathway. Abbreviations: ATGL, adipose triglyceride lipase; C, cancer; FA, fatty acid; HE, hematoxylin-eosin; 5hmC, 5-hydroxymethylcytosine; N, noncancerous tissue; PNPLA2, patatin like phospholipase domain containing 2; TG, triglyceride.