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. 2023 May 24;44(10):2004–2018. doi: 10.1038/s41401-023-01101-x

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a Expression of FGF7 subfamily members in doxorubicin-induced myocardial injury determined by qRT-PCR. b Western blotting analysis of FGF10 protein expression in major viscera after doxorubicin injection. c Western blotting analysis of FGF10 protein expression in the mouse heart after doxorubicin injection. d Immunohistochemical staining of FGF10 in cardiac sections. Top row: ×200 magnification. Bottom row: ×400 magnification. e Statistical analysis of Western blotting presented in c. The results are shown as relative gray value. f Statistical analysis of immunohistochemistry in d. The results are shown as positive staining area per total area of the heart (% Area). GAPDH was used as an internal control. DOXO doxorubicin, FGF10 Fibroblast growth factor 10. ns non-significant; *P < 0.05; **P < 0.01; ****P < 0.0001. Scaler bar: 100 μm.