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. 2023 Sep 21;10:1207219. doi: 10.3389/fcvm.2023.1207219

Table 1.

Clinical characteristics of the study population.

Variables No NOAF group (n = 539) NOAF group (n = 41) p-Value
Age, years 61 (42–78) 64 (45–79) 0.021
Gender (male), n (%) 360 (66.8) 28 (68.3) 0.84
Current smoker, n (%) 216 (40.1) 14 (34.1) 0.51
Alcohol use, n (%) 110 (20.4) 12 (29.3) 0.23
Diabetes mellitus, n (%) 226 (41.9) 25 (61.0) 0.02
Hypertension, n (%) 299 (55.5) 30 (73.2) 0.03
Dyslipidemia on admission, n (%) 188 (34.9) 16 (39.0) 0.61
Previous stroke, n (%) 32 (5.9) 4 (9.8) 0.31
Previous MI, n (%) 34 (6.3) 3 (7.3) 0.74
Previous PCI, n (%) 22 (4.1) 3 (7.3) 0.41
Family history of CAD, n (%) 102 (18.9) 10 (24.4) 0.41
Peripheral vascular disease, n (%) 43 (8.0) 5 (12.2) 0.37
BMI, kg/m2 24.5 ± 3.1 24.7 ± 1.9 0.65
Sleep apnea, n (%) 52 (9.6) 5 (12.2) 0.59
Killip ≥2, n (%) 102 (18.9) 16 (39.0) <0.01
Medication history
ACEI/ARB, n (%) 380 (70.5) 30 (73.2) 0.86
Beta-blocker, n (%) 322 (59.7) 26 (63.4) 0.74
Statins, n (%) 532 (98.7) 40 (97.6) 0.55
Calcium channel blockers, n (%) 141 (26.2) 15 (36.6) 0.15
Diuretics, n (%) 103 (19.1) 9 (22.0) 0.68
Echocardiographic analysis
LA, mm 32.6 ± 4.8 35.7 ± 6.5 <0.01
LVEDV, mm 102.5 ± 22.0 106.8 ± 34.9 0.24
LVESV, mm 59.2 ± 15.6 63.5 ± 28.3 0.12
LVEF (%) 42.5 ± 6.0 42.2 ± 6.8 0.80
Laboratory analysis
NT-proBNP, pg/mL 3,512.8 ± 5,883.3 5,487.7 ± 7,071.3 0.04
CRP, mg/L 7.6 ± 8.0 12.0 ± 7.3 <0.01
Creatine kinase, U/L 88.1 ± 48.4 84.0 ± 25.8 0.59
sST2, ng/mL 48.7 ± 23.6 71.5 ± 34.8 <0.01

MI, myocardial infarction; PCI, percutaneous coronary intervention; CAD, coronary artery disease; BMI, body mass index; ACEI, angiotensin-converting enzyme inhibitors; ARB, angiotensin II receptor blocker, LA, left atrium; LVEDV, left ventricular end diastolic volume; LVESV, left ventricular end systolic volume; LVEF, left ventricular ejection fraction; CRP, C-reactive protein; sST2, soluble ST2.