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. 2023 Sep 29;67:10.29219/fnr.v67.9547. doi: 10.29219/fnr.v67.9547

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© 2023 Zhicui Yao et al.

This is an Open Access article distributed under the terms of the Creative Commons Attribution 4.0 International License, allowing third parties to copy and redistribute the material in any medium or format and to remix, transform, and build upon the material for any purpose, even commercially, provided the original work is properly cited and states its license.

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Fig. 8 — Corn peptides promoted mitochondrial autophagy in HepG2 cells via activating PINK1-Parkin signaling pathway. (A) The protein levels of PINK1 and Parkin in FFAs-induced HepG2 cells. (B) The protein levels of PINK1 in HepG2 cells transferred by siRNA. (C) Oil Red O stain in HepG2 cells transferred by siRNA (magnification, ×200). (D) The intracellular TG content in HepG2 cells transferred by siRNA. (E) The protein levels of Parkin, P62, and ATG7 in HepG2 cells transferred by siRNA. Results were presented as mean ± SD. ^*P < 0.05, ^**P < 0.01, ^***P < 0.001 versus FFAs group (n = 3). FFAs: free fatty acids; CPs: 15 mg/ml corn peptides in control media. The experiment was repeated at least three times.