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. Author manuscript; available in PMC: 2023 Oct 6.
Published in final edited form as: Placenta. 2022 Jun 18;126:54–63. doi: 10.1016/j.placenta.2022.06.004

Table 2.

Placenta microscopic assessment in 1101 participants.

Features of maternal vascular malperfusion
Decidual arteriopathy Muscular hypertrophy Fibrinoid necrosis Fibrinoid necrosis with atherosis
100 (9.1%) 57 (57.0%) 43 (43.0%) 0 (0.0%)
Features of fetal vascular malperfusion
Thrombosis of cord vessels Artery Vein Both
22 (2.0%) 6 (27.3%) 15 (68.2%) 1 (4.5%)
Avascular villi Regional Diffuse
237 (21.5%) 229 (96.6%) 8 (3.4%)
Chorionic plate thrombosis Artery Vein Both Indeterminate
110 (10.0%) 3 (2.7%) 20 (18.2%) 29 (26.4%) 58 (52.7%)
Villous-stromal karyorrhexis Regional Diffuse
293 (26.6%) 268 (91.5%) 25 (8.5%)
Inflammatory response
Chorioamnionitis Acute Chronic Eosinophilic
361 (32.8%) 333 (92.2%) 28 (7.8%) 0 (0.0%)
Subchorion Chorion Chorioamnion Indeterminate
232 (64.3%) 68 (18.8%) 60 (16.6%) 1 (0.3%)
Villitis Acute Chronic specific VUE
249 (22.6%) 2 (0.8%) 7 (2.8%) 240 (96.4%)
Funisitis Perivascular Superficial Both
56 (5.1%) 54 (96.4%) 2 (3.6%) 0 (0.0%)
Vasculitis One vessel Two vessels Three vessels
75 (6.8%) 46 (61.3%) 9 (12.0%) 20 (26.7%)
Artery One Two
33 (44.0%) 13 (39.4%) 20 (60.6%)

VUE villitis of unknown etiology (chronic non-specific).