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Schematic representation of the working model. AAP selectively enhanced P. cinnamivorans, a commensal bacterium with reduced presence in HFD mice to attenuate obesity and the associated dysregulated metabolism. Mechanistically, P. cinnamivorans regulated intestinal lipids metabolism and liver thermogenesis by reducing the proinflammatory response and gut permeability in a JAK-STAT signaling-related manner.
