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. 2023 Aug 2;31(10):3067–3083. doi: 10.1016/j.ymthe.2023.07.024

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miR-26a-5p derived from MSC-EVs inhibited kidney inflammation and fibrosis in vivo

(A) PAS staining and immunohistochemical staining (ZEB2, TGF-β1) of IRI-3 d kidney tissue +/− miR-26a-5p inhibitor treatment. (B) Flow cytometry plots showing the proportion of Th17 cells in IRI-3 d kidney tissue +/− miR-26a-5p inhibitor treatment. (C) PAS, Masson’s trichome, and Sirius red staining indicating fibrosis levels in IRI-7 d kidney tissue +/− miR-26a-5p inhibitor treatment. (D) Representative images of immunohistochemical staining showing the levels of ZEB2, TGF-β1, and E-cadherin in IRI-7 d kidney tissue +/− miR-26a-5p inhibitor treatment.