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. 2023 Oct 4;52(5):111. doi: 10.3892/ijmm.2023.5314

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Copyright: © Zheng et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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PPAR, STAT3 and PI3K/Akt signaling pathways and targets of inhibitors against neuroinflammation in AD. PPAR, peroxisome proliferator-activated receptor; AD, Alzheimer's disease; 1, berberine; 2, rice bran extract; 3, telmisartan; 4, bis(ethylmaltolato)oxidovanadium; 5, platycodigenin; 6, (E)-2, 4-bis(p-hydroxyphenyl)-2-butenal; 7, (E)-2-methoxy-4-(3-(4-methoxyphenyl) prop-1-en-1-yl) phenol; 8, astaxanthin; 9, stattic; 10, protosappanin A; 11, curcumin; 12, Ent-Sauchinone; 13, sorafenib; 14, DHCR24 (3-β-hydroxysteroid-Δ-24-reductase); 15, sulforaphene; Aβ, amyloid β; LPS, lipopolysaccharide; IKK, IκB kinase; JAK2, Janus kinase; GSK-3β, glycogen synthase kinase-3β; PTEN, phosphate and tensin homolog deleted on chromosome 10.