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. 2023 Oct 11;16(10):dmm049326. doi: 10.1242/dmm.049326

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© 2023. Published by The Company of Biologists Ltd

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (https://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution and reproduction in any medium provided that the original work is properly attributed.

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Fig. 5. — pkd1l1 mutant zebrafish have abnormal liver biochemistry. Whole livers were isolated from 6-week-old zebrafish and homogenized for protein assays. (A) Total liver protein concentration showed no significant differences between wild-type and pkd1l1 mutant livers (n=16). (B,C) ALT (B) and GGT (C) enzyme assays showed a significant increase in enzyme activity in pkd1l1 mutant livers compared to that in wild-type livers (n=15). Graphs show the mean±s.e.m. ns, not significant; ***P<0.001; ****P<0.0001 (unpaired two-tailed Student's t-test).