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. 2023 Sep 13;80(11):2386–2396. doi: 10.1161/HYPERTENSIONAHA.123.21645

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© 2023 The Authors.

Hypertension is published on behalf of the American Heart Association, Inc., by Wolters Kluwer Health, Inc. This is an open access article under the terms of the Creative Commons Attribution Non-Commercial-NoDerivs License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited, the use is noncommercial, and no modifications or adaptations are made.

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Figure 2. — Cellular changes induced by Rosiglitazone. A, Bubble plots visualizing the top 15 enriched gene ontology (GO) functions of differentially expressed gene isoforms related to Rosiglitazone in BeWo cells under hypoxia/reoxygenation (H/R) conditions, with a focus on biological processes (BP). B, Significantly increased expression of 2 HO1 (heme oxygenase 1) isoforms (ENST00000216117; HMOX1-201 and ENST00000678411; HMOX1-206) in BeWo cells exposed to Rosiglitazone under H/R conditions.