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. 2023 Oct 4;14:1267091. doi: 10.3389/fimmu.2023.1267091

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Copyright © 2023 Xiang, Zhang, Jiang, Su and Shi

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The pathogenesis of SLE. The appearance of NETosis in neutrophils promotes the secretion of IFN-α/β by pDC cells to stimulate the body’s innate immunity and adaptive immunity, while the frontal interaction between inflammatory cells and lymphoid T and B cells results in the production of a large number of inflammatory factors. APCs, antigen-presenting cells. TCR, T cell receptor. BCR, B cell receptor. BAFFR, B-cell activating factor receptor. CRP, C-reactive protein; ESR, erythrocyte sedimentation rate.