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. 2023 Oct 18;13:17736. doi: 10.1038/s41598-023-44836-7

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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PD ameliorated oxidative stress by up-regulating the Nrf-2 signaling pathway in CRKP-infected A549 cells. CRKP-induced cellular injury induced by PD and ML385 was detected by LDH (A). The cells were incubated with CRKP and PD for 12 h and the concentration of SOD (B), MPO (C) were assessed by corresponding kits. A representative result of the protein expression of Nrf-2 among all groups was analyzed by western blot, and a statistical result was calculated (D-E). ^##P < 0.01 vs. the control group; ^*P < 0.05 and ^**P < 0.01 vs. the CRKP alone group.