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. 2023 Oct 18;13:17803. doi: 10.1038/s41598-023-45017-2

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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The protective effect of CGA pretreatment on MIRI in mice. (a) Evans blue was injected via the aorta after myocardial ischemia reperfusion. Blue area: noninfarcted area, red area: infarcted area. (b,c) The proportion of the infarct area in the CGA + I/R group was far lower than that in the I/R group, and the infarcted tissue (white area) was identified by TTC staining. (d,e) The effect of CGA on LDH and CK-MB activity in I/R-injured mice. The activity of LDH and CK-MB in mouse serum were measured by an automatic biochemical analyzer system. (f) The effect of CGA on content of cTnT in I/R-injured mice. The content of cTnT in mouse serum was measured by an automatic biochemical analyzer and electroluminescence detection system. (g) Pathological changes of myocardial tissue were observed by HE staining. Representative images were captured using an optical microscope (×400 magnification). The data are expressed as the mean ± SD. n = 15 mice in each group. *P < 0.05 vs. the sham group, ^#P < 0.05 vs. the I/R group.