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. 2023 Oct 20;18:115. doi: 10.1186/s13000-023-01403-y

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Pathological features of ANHH. (A) Macroscopic and (B) microscopic examinations (H&E) and (C) insulin immunostaining reveal no mass lesion in the sections of the resected pancreas. (D) Ductuloinsular complex. H&E. (E) Multiple β-cells with an enlarged and hyperchromatic nucleus and abundant clear cytoplasm. H&E. (F) Macronucleoli in β-cells. H&E. (G-J) Immunostaining. Islets with normal spatial distribution of the various cell types. (G) Insulin, (H) Glucagon, (I) Somatostatin, and (J) Pancreatic polypeptide staining. (K) Ki-67 immunostaining reveals no proliferative activity of the islet cells. Scale bars in B, C, G-K are 250 μm; the scale bar in D is 100 μm; the scale bar in E is 50 μm; and the scale bar in F is 25 μm