Skip to main content
. 2023 Oct 9;13:1274048. doi: 10.3389/fonc.2023.1274048

Table 1.

Summary of the mechanisms of DC activation by natural polysaccharides.

Name Phenotypic maturation Membrane target Downstream signalling T cell activation Cytokine production Anti-tumor effect References
POL-P3b ↑CD40, CD80, CD86, MHC-II TLR4 NF-κB ↑proliferation of CD4+ and CD8+ T cells and CD4+/CD8+ ↑IFN-γ, IL-12p70, TNF-α, and IL-4 POL-P3b-treated DC vaccine can improve tumor inhibition rate and inhibit lung metastasis (78)
APS ↑CD206, CD80, CD86 Not tested NF-κB ↑proliferation of CD45+CD8+ T cells Not tested APS suppressed tumorigenicity and metastasis in syngeneic C57BL/6 mice, and potentiated anticancer effect of cisplatin In vivo (79)
LNT ↑CD86 Not tested Not tested Not tested ↑IL-6, IL-10, and TGF-β1 The survival period of Colon-26-bearing mice treated with S-1 + LNT was significantly more prolonged than that of mice treated with S-1 alone (80)
GLP ↑CD40, CD80, CD86, MHC-II TLR4 Not tested ↑proliferation of CD4+/CD44+ memory T cells ↑mRNA expression of IL-6, IL-12, IL-1β, TNF-α, and IFN-γ GLP exhibited strong inhibitory effects on 4T1 tumor growth and pulmonary metastasis when combined with doxorubicin (81)
NGP ↑CD40, CD80, CD83, CD86, MHC-II Not tested Not tested ↑proliferation of T cells ↑IL-12p70, TNF-α Not tested (82)
APS-AuNP ↑CD40, CD80, CD86, MHC-II Not tested Not tested ↑proliferation of CD4+ T cells and CD8+ T cells ↑IL-6, IL-1β, IFN-γ, and TNF-α The inhibitory rate of APS-AuNP against 4T1 primary tumor growth and pulmonary metastasis in mice was higher than paclitaxel-treated group (83)
POL-P3b ↑CD40, CD80, CD86, MHC-I, MHC-II TLR4 PI3K/AKT (↑phosphorylation of AKT) Not tested Not tested The tumor weight of U14-bearing mice in the POL-P3b group was significantly lower than that of the model group (84)
Angelan ↑MHC-I, MHC-II, CD80, CD86 Not tested MAPK (↑ phosphorylation of ERK, JNK, and p38);
NF-κB (↑nuclear translocation of p50, p65, RelB, and c-Rel)
Not tested ↑IL-12, CCL19, CXCL12 Angelan-treated mature DC more effectively inhibited B16F10 tumor growth than immature DCs in syngenic murine tumor model (85)
AC hmwPS ↑CD40, CD80, CD86, MHC-I, MHC-II TLR2 and TLR4 MAPK (↑ phosphorylation of ERK, JNK, and p38);
NF-κB (↑nuclear translocation of p65)
↑proliferation of CD8+ T cells;
↑Th1 differentiation
↑TNF-α, IL-6, and IL-12 In a mouse tumor model, AC hmwPSs enhanced the anti-tumor efficacy of the HER-2/neu DNA vaccine by facilitating specific Th1 responses (86)
YM-2A ↑CD80, CD86 Dectin-1 Dectin-1-dependent pathway ↑proliferation of CD4+ and CD8+ T cells ↑IFN-γ YM-2A-treated TAA-loaded DC vaccine significantly reduced tumor growth and improved survival in two murine tumor models, CT-26 tumor-bearing BALB/c mice and B16 melanoma-bearing C57BL/6 mice (87)
LBP ↑CD80, CD86 Not tested ↑expression of Notch, Jagged, Hes1, and Hes5 ↑proportion of CD3+CD8+ T cells ↑IL-12;
↓IL-10, TGF-β
Administration of LBP Strengthened the Cytotoxicity of DC-Mediated CTLs on Colon Cancer Cell CT26-WT (88)
PLP ↑CD80, CD86, MHC-II Not tested Not tested ↑proportion of CD3+CD8+ T cells ↑IL-12p70, TNF-α, IL-1β, and IL-6 TAXOL+PLP significantly inhibited 4T1 tumor growth, compared with the control group and TAXOL group (89)
RGP ↑CD40, CD80, CD86, MHC-I, MHC-II TLR4 Not tested ↑proportion of OT-I and OT-II T cells ↑IL-6, IL-12p40, TNF-α The combination of RGP and Ag effectively inhibited the growth of CT57 tumors and B6 melanoma in BLAB/c and C26BL/16 mice (90)
GLP ↑CD40, CD80, CD86, and MHC-II Not tested Not tested ↑proportion of CD3+CD8+ T cells ↑IL-10 and IL-12p70 GLP significantly reduced the tumor volume of glioma-bearing rats to 101.93 ± 53.58 mm3 and 113.56 ± 39.76 mm3, compared to 162.99 ± 48.34 mm3 in the control group (91)
PS-F2 ↑CD40, CD80, CD86, and MHC-II Not tested Not tested ↑Th1 differentiation ↑TNF-α, IL-12/IL-23 p40, IL-6, and
IL-10
Mice immunized with PS-F2 and OVA were almost completely protected from MO5 tumor challenge (92)

↑, increases; ↓, decreases.