Studies evaluating EEG to detect NCSz after acute brain injury

Authors	Design	Population	N	Findings
TBI 0–33 % NCSz
Steudel et al. [218]	R CS		50	8 % on routineEEGs
Vespa et al. [107]	P CS		94	22 % NCSz
Ronne-Engstrom et al. [111]	R CS		70	33 % NCSz
Olivecrona et al. [112]	P CS		47	0 % NCSz, 8.5 % clinical seizures pre EEG
SAH 3–31 % NCsz
Dennis et al. [240]	R CS		233	3 % (31 % or 8 of 26 with EEG)
Claassen et al. [214]	R CS		116	15 % NCSz, 11 % NCSE
Little et al. [241]	R CS		389	3 % (but only very small number got EEG)
ICH 18–28 %
Vespa et al. [88]	Pr CS		109	28 % NCSz (only one convulsive)
Claassen et al. [87]	R CS		102	18 % NCSz (only one convulsive), 7 % NCSE
PRES
Kozak et al. [119]	R CS		10	PRES presented in all cases with SE
CNS infection 33 % NCsz
Carrera et al. [116]	R CS	64 % viral	42	33 % NCSz
AIS 2 % NCSz
Carrera et al. [124]	P CS	AIS stroke unit	100	2 % NCSz
Mixed neuro ICU populations
Jordan et al. [242]	R CS	Mixed NICU		NCSz 34 %
Claassen et al. [4]	R CS	ICU/ward	570	11 % NCSz, 20 % NCSE
Pandian et al. [3]	R CS	Mixed NICU	105	No denominator
Amantini et al. [113]	P CS	TBI, ICH, SAH	68	3 % NCSz
Drislane et al. [243]	R CS	All NCSE	91	No denominator
Mecarelli et al. [115]	P CS	ICH/SAH/AIS	232	6 % NCSz, 4 %NCSE (spot EEG < 24 h)

R retrospective, P prospective, CS case series, NCSz nonconvulsive seizures, NCSE nonconvulsive status epilepticus, ICH intracerebral hemorrhage, SAH subarachnoid hemorrhage, AIS acute ischemic stroke, TBI traumatic brain injury, CNS central nervous system