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. 2023 Aug 16;149(16):14641–14655. doi: 10.1007/s00432-023-05256-2

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 8 — The effects of ACC1 and ACLY on GPR176-mediated chemoresistance and lipogenesis. Either ACC1 and ACLY was overexpressed in shGPR176 transfectants of KYSE-150 cells, as confirmed by western blotting (A). After 5-FU or taxol treatment, KYSE-150 cells and transfectants were analysed using CCK-8 (B) and Nile red staining (C). KYSE, KYSE-150 cells; **p < 0.01; ***p < 0.001