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. 2023 Oct 26;14:6797. doi: 10.1038/s41467-023-42273-8

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 10 — a Cntn1 interacts with and is redundantly recruited to the AIS through interactions with both NrCAM and Nfasc. b Loss of AIS Cntn1 (orange at AIS) from cerebellar Purkinje neurons disrupts basket cell innervation of the AIS and formation of pinceau synapses. c Loss of AIS Cntn1 (orange) from Pyramidal neurons results in reduced innervation of AIS by Chandelier cells (ChC) and reduced numbers of AIS inhibitory synapses. Created with Biorender.com.