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. 2023 Oct 17;38(5):482–492. doi: 10.3803/EnM.2023.1776

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Copyright © 2023 Korean Endocrine Society

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 2. — Comparative analysis of ameliorative effects caused by taurine and its derivatives. Top to bottom N-propionyl N-cholrotaurine, taurolidine (TRD), 2 (D-glucopyranosyl) ethyl amino, N-(D-ribopyranosyl) taurine sodium salt, N (D-lyxopyronosyl) taurine sodium salt, N- (D-ribopyranosyl) taurine sodium salt, 1-deoxy (2 sulphoethylamine)-D-fructose, 1.4.5 oxathiaziane 4,4-dioxide (OTD), N-(aldopyranosyl) taurine, 4 nitrophenyl 1-6-deoxy-6-[2 sulphoethyl amino], β-D-galactopyranoside, N-(β-D-glucopyranosyl) taurine sodium salt, tauronustine, acamprostate, taltrimide, tauroursodeoxycholic acid sodium salt (TUCDA), N-phenoyl taurine, chloramine, N-(β-dxylopyranosyl) taurine sodium salt, N-(β-D-arabinopyranosyl) taurine sodium salt, taurine.