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. 2023 Oct 31;21:237. doi: 10.1186/s12915-023-01697-w

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 9 — A working model of VdZFP1 and VdZFP2 during microsclerotia development of Verticillium dahliae. The cytoplasm-localized C2H2-type zinc finger proteins VdZFP1 and VdZFP2 enter the nucleus by sensing environmental signals and are regulated by the MAPK signaling pathway to promote microsclerotia development and melanization by interacting with VdCmr1 on the cell wall