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. 2023 Nov 1;9:405. doi: 10.1038/s41420-023-01698-2

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 8 — A–C The expression of YTHDF2 and C1qA in Farage/R cells with YTHDF2 knockdown (R-shYTHDF2) and in control cells (R-shCtrl). D The level of m⁶A-modified C1qA. E Cell survival rates and the IC50 value of Rituximab. F The proliferation of cells being treated with 20 μg/mL Rituximab was detected by the colony formation assay. G C3 chains expressed during treatment with 20 μg/mL Rituximab were detected by western blotting. H The proportions of apoptotic cells during treatment with 20 μg/mL Rituximab were detected by flow cytometry. ***p < 0.001.