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. 2023 Aug 8;17(11):2472–2490. doi: 10.1002/1878-0261.13499

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© 2023 The Authors. Molecular Oncology published by John Wiley & Sons Ltd on behalf of Federation of European Biochemical Societies.

This is an open access article under the terms of the http://creativecommons.org/licenses/by/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Fig. 6 — The novel genes of MCM4 and CXCL6 driving the dysfunctional cancer hallmarks in TCGA‐06‐0648. (A) The dysfunctional cancer hallmarks in TCGA‐06‐0648. (B) The correlation between dysfunctional scores and enrichment scores driven by driver genes in GBM individual TCGA‐06‐0648. P was calculated by R function cor.test(). (C) The cumulative contributions of MCM4 and CXCL6 on the dysfunction of cancer hallmarks. (D) The dysfunction of cancer hallmarks driven by MCM4 and CXCL6. (E) High expression of MCM4 were significantly associated with poor GBM prognosis. P was calculated by Log‐rank test.