Skip to main content
. 2023 Oct 19;14:1249436. doi: 10.3389/fimmu.2023.1249436

Figure 1.

Figure 1

Loss of the gonadal heterotypic heteromeric gap junction subunit gene inx-14 causes enhanced resistance to P. aeruginosa PA14 infection. (A – F) PA14 survival assays of wild-type and gap junction subunit gene mutants inx-8(gk42) (A), inx-9(gk293) (B), inx-14(ag17) (C), inx-19(ky634) (D), inx-21(ok2514) (E), and inx-22(tm1661) (F). (G–V) Survival assays of wild-type and gap junction subunit gene mutants che-7(ok3373) (G), eat-5(ad1402) (H), unc-7(e5) (I), unc-9(e101) (J), inx-1(tm3524) (K), inx-2(ok375) (L), inx-5(ok1053) (M), inx-6(rr5) (N), inx-7(ok2319) (O), inx-10(ok2714) (P), inx-11(ok2783) (Q), inx-15(tm3394) (R), inx-16(ox144) (S), inx-17(tm3292) (T), inx-18(ok2454) (U), and inx-20(ok181) (V). All experiments were repeated at least three times. Statistical significance was determined by log-rank test. *P < 0.05; **P < 0.01; ***P < 0.001; n.s., not significant. The exact P values of statistics for all survival assays are listed in Supplementary Table S1 .