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. 2023 Nov 2;23:259. doi: 10.1186/s12935-023-03091-6

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — The potential mechanism of SLC7A11 in ACC and disulfidptosis development A, B GO and KEGG enrichment analysis of 686 blue module genes. C The volcano plot shows all genes related to SLC7A11,and we have highlighted 12 prominent genes among them. D, E Top 50 SLC7A11-related genes with positive/negative correlations. F, G, H GO enrichment analysis (BP, CC, MF) of these genes in the LinkedOmics database