Figure 1. An overview of the endocannabinoid pathway and molecules tested in this study.

The cellular apoptosis cascade is mediated by a multitude of molecules and receptor-ligand interactions. Fatty acid amide hydrolase (FAAH) has been documented to inhibit the apoptosis pathway. Thus, FAAH inhibitors, PF750 and URB597, have promising potential in inducing apoptosis in breast cancer cells. Moreover, cannabinoids, such as AEA and PEA, promote apoptosis, serving as another potential therapeutic agent in cancer cells.