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. 2023 Nov 10;42:296. doi: 10.1186/s13046-023-02875-y

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — lncRNA AC016727.1 promotes tumor proliferation, invasive migration, and aerobic glycolytic progression via BACH1. a-c BACH1 enhanced A549 and H1299 proliferation following lncRNA AC016727.1 knockdown. Quantitative analysis is presented on the right. d BACH1 inhibited A549 and H1299 cell apoptosis following lncRNA AC016727.1 knockdown. Quantitative analysis is presented on the right. e BACH1 promoted the migratory and invasive capacities of lncRNA AC016727.1-overexpressing A549 and H1299 cells. Quantitative analysis is presented on the right. f Following overexpression or knockdown of the lncRNA AC016727.1, EMT and cellular glycolysis marker proteins were identified to assess the impact of BACH1 on EMT in the A549 cells. Quantitative analysis is presented on the right. g Following overexpression or silencing of the lncRNA AC016727.1, EMT and cellular glycolysis marker proteins were detected to assess the impact of BACH1 on EMT in the H1299 cells. Quantitative analysis is presented on the right