Table 1.

The mechanism of lncRNA H19 in different renal cells in diabetic kidney diseases.

Expression	Samples	Targets	Functions	References
up	Mesangial cells	miR-143-3p, IL-6, TNF-α, TGF-β1, Col. IV, and FN	Inflammation, Proliferation, Fibrosis	(111)
up	Serum	IL-6, TNF-α	Fibrosis	(112, 113)
up	STZ-mice, podocyte	miR-29a, TGFβ, Smad3, FSP-1, CD31	Fibrosis, EndMT	(114, 115)
up	HK-2 cells	miR-17, α-SMA, FN, Col. IV, and Col. I	Fibrosis, EMT	(116)
up	Mesangial cell	miR-129, HMGB1, Nrf2	Ferroptosis	(117)
up	podocytes	NLRP3	Pyroptosis	(118)
up	podocytes	ATG7	Autophagy	(119)
up	serum	Beclin-1	Autophagy	(120)
up	Tubular epithelial cells	DIARS3, mTOR	Autophagy	(121)
up	CIHP‐1, HEK 293 cells	miR-675, VDR, EGR1	EMT, Fibrosis,	(122)
up	Diabetic mice	MFN-2	Mitochondrial dysfunction	(123)
up	DKD rats, glomerular endothelial cells	Akt/eNOS	GEC damage	(124–126)