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. 2023 Jun 2;396(12):3595–3603. doi: 10.1007/s00210-023-02553-y

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Predicted molecular mechanism of action of BBR LCNP treatment on A549 cells. A The normal state of WNT/β-catenin pathway is shown, where binding of WNT molecules leads to the activation of β-catenin resulting in tumorigenesis. B Inhibition of AXIN1 and β-catenin by BBR LCNPs is shown