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. 2023 Oct 6;20(7):1145–1159. doi: 10.1007/s13770-023-00585-2

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© Korean Tissue Engineering and Regenerative Medicine Society 2023. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law.

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Fig. 3 — miR-16-5p is targeted by circSPEF2. A Map of potential binding sites of circSPEF2 and miR-16-5p; B Ago2 enrichment level of circSPEF2 and miR-16-5p in SU-DHL4 cells; C RT-qPCR checked miR-16-5p; D Dual luciferase reporter assay verified the relationship between circSPEF2 and miR-16-5p; E After transfection of oe-circSPEF2 into SU-DHL4 cells, RT-qPCR checked miR-16-5p; F, G RT-qPCR checked miR-16-5p in tissues of DLBCL patients and cell lines. Data were expressed as mean ± SD (N = 3). p < 0.05