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. 2023 Oct 27;12(10):2030–2039. doi: 10.21037/tlcr-23-449

Table 2. Treatment of patients with KRAS mutation (N=66).

Factors Number (%)
1st-line chemotherapy
   Taxanes-based 8 (12.1)
   Pemetrexed-based 48 (72.7)
   Gemcitabine-based 3 (4.5)
   No 7 (10.6)
1st-line angiogenesis inhibitors
   Yes 28 (42.4)
   No 38 (57.6)
1st-line immune therapy
   Yes 10 (15.2)
   No 56 (84.8)
2nd-line therapy
   Yes 37 (56.1)
   No 27 (40.9)
   Unknown 2 (3.0)
2nd-line therapy
   Taxanes-based 20 (30.3)
   Pemetrexed-based 8 (12.1)
   Gemcitabine-based 3 (4.5)
   Other 6 (9.1)
   No 29 (43.9)
2nd-line angiogenesis inhibitors
   Yes 9 (13.6)
   No 28 (42.4)
2nd-line immune therapy
   Yes 9 (13.6)
   No 28 (42.4)
3rd-line therapy
   Yes 17 (25.8)
   No 49 (74.2)
4th-line therapy
   Yes 8 (12.1)
   No 58 (87.9)
5th-line therapy
   Yes 3 (4.5)
   No 63 (95.5)
Posterior line therapy 28 (100.0)
   Chemotherapy 8 (28.6)
   Angiogenesis inhibitors 5 (17.9)
   Chemotherapy + angiogenesis inhibitors 5 (17.9)
   Chemotherapy + immune therapy 2 (7.1)
   Angiogenesis inhibitors + immune therapy 5 (17.9)
   TKI 2 (7.1)
   Chemotherapy + TKI 1 (3.6)

Posterior line therapy: 3rd-line, 4th-line and 5th-line therapy. KRAS, Kirsten rat sarcoma viral oncogene homolog; TKI, tyrosine-kinase inhibitor.