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. 2023 Aug 14;8(11):2333–2344. doi: 10.1016/j.ekir.2023.07.037

Table 3.

Proportion of AKI risk factors

AKI risk factor, n (%) Entire cohort (N = 314) DI-AKI (n = 271) Not DI-AKI (n = 43) P-value
Hyperglycemia 144 (45.86) 131 (48.34) 13 (30.23) 0.03
Severe infection or sepsis 56 (17.83) 50 (18.45) 6 (13.95) 0.67
Extracellular fluid loss 41 (13.06) 38 (14.02) 3 (6.98) 0.33
Red blood cells transfusion 39 (12.42) 30 (11.07) 9 (20.93) 0.08
Intravascular fluid loss 37 (11.78) 34 (12.55) 3 (6.98) 0.44
Other procedures 31 (9.9) 29 (10.7) 2 (4.7) 0.28
Liver disease 30 (9.55) 24 (8.86) 6 (13.95) 0.27
Anesthetic agent 28 (8.92) 22 (8.12) 6 (13.95) 0.24
Cardiac failure 27 (8.6) 25 (9.23) 2 (4.65) 0.56
Increased vascular capacity 14 (4.46) 9 (3.32) 5 (11.63) 0.03
Cardiac surgery 7 (2.23) 3 (1.11) 4 (9.3) <0.01
Vascular surgery 4 (1.27) 3 (1.11) 1 (2.33) 0.45
Hepatorenal syndrome 3 (0.96) 3 (1.11) 0 (0) 1
Hemorrhage 2 (0.64) 2 (0.74) 0 (0) 1

AKI, acute kidney injury; DI-AKI, drug-induced acute kidney injury; RBC, red blood cell.

AKI risk factors were considered to be present if they were recorded in the 72 hours preceding AKI onset.

Increased vascular capacity was defined as clinical events leading to reduced blood perfusion (e.g., mean arterial pressure <65 mm Hg, sepsis). Hyperglycemia was defined as blood sugar >110 mg/dl or the patient is on insulin.