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. 2023 Nov 20;14:7538. doi: 10.1038/s41467-023-43178-2

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a Association between captured subgenome variations and polyploidy-promoted diversity. The captured variations are reflected by the differences between subgenomes within the same cell. The polyploidy-promoted diversity was evaluated on the basis of population diversity (π). b Enrichment of the binding of trans-factors to the four regions shown in panel (a). c Enrichment of epigenetic marks in subgenome-specific regions associated with high population diversity. The epigenetic data were generated using Chinese Spring (CS) and JW1. d Genomic tracks illustrating the genomic distribution of population diversity (π), subgenome-specific regions, and the loci of LHP1-binding sites and H3K27me3.