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. 2023 Sep 22;101(11):1449–1464. doi: 10.1007/s00109-023-02369-6

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — mRNA seq analysis of gemcitabine-resistant pancreatic cancer cells. A Scatter plots indicate the distribution of genes from Mia Con and Mia GR cells. B DAVID-based gene ontology and C KEGG pathway analysis of mRNA seq results from the Mia GR cells. D Gene Set Enrichment Analysis (GSEA) determined significant enrichment for glucose metabolic process, TNF signaling via NF-κB and NF-κB pathway in Mia GR and Mia Con cells.